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IgE

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Cat# I1900-02C-500ug

Size : 500ug

Brand : US Biological


I1900-02C IgE

Clone Type
Monoclonal
Host
rat
Source
mouse
Swiss Prot
P06336
Isotype
IgG2a
Grade
Affinity Purified
Applications
E
Crossreactivity
Mo
Shipping Temp
Blue Ice
Storage Temp
-20°C

Applications:
Suitable for use in ELISA. Other applications not tested.

Recommended Dilution:
Optimal dilutions to be determined by the researcher.

Affinity Constant:
2x10e9 L/mole.

Storage and Stability:
May be stored at 4°C for short-term only. Aliquot to avoid repeated freezing and thawing. Store at -20°C. Aliquots are stable for 12 months after receipt. For maximum recovery of product, centrifuge the original vial after thawing and prior to removing the cap.

Applications
Product Type: Mab|Isotype: IgG2a|Clone No: LO-ME-2|Host: rat|Source: mouse|Concentration: ~1mg/ml|Form: Supplied as a liquid in PBS, 0.09% sodium azide.|Purity: Purified by affinity chromatography from tissue culture supernatant.|Immunogen: Purified IgE from Balb/c mice.|Specificity: Recognizes the epsilon heavy chain of mouse IgE. No cross reactivity is seen with other mouse immunoglobulins, as tested in an ELISA.||Important Note: This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications without the expressed written authorization of United States Biological.
Immunogen
Purified IgE from Balb/c mice.
Form
Supplied as a liquid in PBS, 0.09% sodium azide.
Purity
Purified by affinity chromatography from tissue culture supernatant.
Specificity
Recognizes the epsilon heavy chain of mouse IgE. No cross reactivity is seen with other mouse immunoglobulins, as tested in an ELISA.
References
1. Kikuchi, Y. et al. (2006) Crucial commitment of proteolytic activity of a purified recombinant major house dust mite allergen Der p1 to sensitization toward IgE and IgG responses. J Immunol. 177(3): 1609-17.|2. Peebles Rs, J.r. et al. (2005) Allergen-induced airway hyperresponsiveness mediated by cyclooxygenase inhibition is not dependent on 5-lipoxygenase or IL-5, but is IL-13 dependent. J Immunol. 175(12): 8253-9.|3. Hashimoto, K. et al. (2005) Cyclooxygenase inhibition augments allergic inflammation through CD4-dependent, STAT6-independent mechanisms. J Immunol. 174(1): 525-32.|4. Peebles Rs, J.r. et al. (2000) Cyclooxygenase inhibition increases interleukin 5 and interleukin 13 production and airway hyperresponsiveness in allergic mice. Am J Respir Crit Care Med. 162(2 Pt 1): 676-81.